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Enta has important roles in pregnancy and parturition and is a major circulating hormone during pregnancy in all mammalian species acting on the pubic symphysis, cervix, uterus, vagina, and mammary glands. It also is responsible for many of the cardiovascular changes that occur during pregnancy (Debrah et al., 2006; Conrad, 2011). Relaxin causes increased flexibility and elasticity of the interpub
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Enta has important roles in pregnancy and parturition and is a major circulating hormone during pregnancy in all mammalian species acting on the pubic symphysis, cervix, uterus, vagina, and mammary glands. It also is responsible for many of the cardiovascular changes that occur during pregnancy (Debrah et al., 2006; Conrad, 2011). Relaxin causes increased flexibility and elasticity of the interpub
1
Enta has important roles in pregnancy and parturition and is a major circulating hormone during pregnancy in all mammalian species acting on the pubic symphysis, cervix, uterus, vagina, and mammary glands. It also is responsible for many of the cardiovascular changes that occur during pregnancy (Debrah et al., 2006; Conrad, 2011). Relaxin causes increased flexibility and elasticity of the interpub
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Ls (Bathgate et al., 2006c), and these effects are impaired in both relaxin-deficient rats (Burger and Sherwood, 1998) and relaxin knockout mice (Zhao et al., 1999; Bathgate et al., 2006c). In humans, although relaxin levels increase during cervical ripening, this still occurs after embryo transfer where circulating relaxin levels are undetectable (Eddie et al., 1990a). In clinical trials, althoug
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Ls (Bathgate et al., 2006c), and these effects are impaired in both relaxin-deficient rats (Burger and Sherwood, 1998) and relaxin knockout mice (Zhao et al., 1999; Bathgate et al., 2006c). In humans, although relaxin levels increase during cervical ripening, this still occurs after embryo transfer where circulating relaxin levels are undetectable (Eddie et al., 1990a). In clinical trials, althoug
1
Ls (Bathgate et al., 2006c), and these effects are impaired in both relaxin-deficient rats (Burger and Sherwood, 1998) and relaxin knockout mice (Zhao et al., 1999; Bathgate et al., 2006c). In humans, although relaxin levels increase during cervical ripening, this still occurs after embryo transfer where circulating relaxin levels are undetectable (Eddie et al., 1990a). In clinical trials, althoug
1
Ls (Bathgate et al., 2006c), and these effects are impaired in both relaxin-deficient rats (Burger and Sherwood, 1998) and relaxin knockout mice (Zhao et al., 1999; Bathgate et al., 2006c). In humans, although relaxin levels increase during cervical ripening, this still occurs after embryo transfer where circulating relaxin levels are undetectable (Eddie et al., 1990a). In clinical trials, althoug
1
Ls (Bathgate et al., 2006c), and these effects are impaired in both relaxin-deficient rats (Burger and Sherwood, 1998) and relaxin knockout mice (Zhao et al., 1999; Bathgate et al., 2006c). In humans, although relaxin levels increase during cervical ripening, this still occurs after embryo transfer where circulating relaxin levels are undetectable (Eddie et al., 1990a). In clinical trials, althoug